Differential regulation of GLUT-1 and GLUT-4 glucose transporter mRNA levels in 3T3-L1 adipocytes
Author:
Affiliation:
1. Sandoz Research Institute, East Hanover, New Jersey 07936, U.S.A.
Publisher
Portland Press Ltd.
Subject
Biochemistry
Link
https://portlandpress.com/biochemsoctrans/article-pdf/18/6/1247/514331/bst0181247.pdf
Cited by 6 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Acute impairment of insulin signalling by dexamethasone in primary cultured rat skeletal myocytes;Molecular and Cellular Biochemistry;2006-10-28
2. Insulin can enhance GLUT4 gene expression in 3T3-F442A cells and this effect is mimicked by vanadate but counteracted by cAMP and high glucose – potential implications for insulin resistance;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2001-02
3. Dexamethasone-induced insulin resistance in 3T3-L1 adipocytes is due to inhibition of glucose transport rather than insulin signal transduction.;Diabetes;2000-10-01
4. THE OXAZOLIDINEDIONE CP-92,768-2 PARTIALLY PROTECTS INSULIN-RECEPTOR SUBSTRATE-1 FROM DEXAMETHASONE DOWN-REGULATION IN 3T3-L1 ADIPOCYTES;ENDOCRINOLOGY;1995
5. Dexamethasone down-regulation of insulin receptor substrate-1 in 3T3-L1 adipocytes.;Journal of Biological Chemistry;1994-01
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