CaMKIIαB Mediates a Survival Response in Retinal Ganglion Cells Subjected to a Glutamate Stimulus
Author:
Affiliation:
1. From the Departments of Anatomical Sciences and Neurobiology and
2. From the Departments of Anatomical Sciences and Neurobiology and2Ophthalmology and Visual Sciences, University of Louisville School of Medicine, Louisville, Kentucky.
Publisher
Association for Research in Vision and Ophthalmology (ARVO)
Subject
General Medicine
Cited by 22 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Unveiling the role of CaMKII in retinal degeneration: from biological mechanism to therapeutic strategies;Cell & Bioscience;2024-05-09
2. Ca2+/Calmodulin-Dependent Protein Kinase II Enhances Retinal Ganglion Cell Survival But Suppresses Axon Regeneration after Optic Nerve Injury;eneuro;2024-03
3. Is CaMKII friend or foe for cell apoptosis in eye?: A narrative review;Medicine;2023-12-01
4. Retinal Glutamate Neurotransmission: From Physiology to Pathophysiological Mechanisms of Retinal Ganglion Cell Degeneration;Life;2022-04-25
5. The cGMP system in normal and degenerating mouse neuroretina: New proteins with cGMP interaction potential identified by a proteomics approach;Journal of Neurochemistry;2020-12-05
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