Tetraspanins CD9 and CD81 function to prevent the fusion of mononuclear phagocytes

Author:

Takeda Yoshito1,Tachibana Isao1,Miyado Kenji2,Kobayashi Masatoshi2,Miyazaki Toru3,Funakoshi Toshiki1,Kimura Hiromi1,Yamane Hiroyuki1,Saito Yoshiyuki1,Goto Hiroyuki1,Yoneda Tsutomu1,Yoshida Mitsuhiro1,Kumagai Toru1,Osaki Tadashi1,Hayashi Seiji1,Kawase Ichiro1,Mekada Eisuke2

Affiliation:

1. Department of Molecular Medicine, Osaka University Graduate School of Medicine, Osaka 565-0871, Japan

2. Research Institute for Microbial Disease, Osaka University, Osaka 565-0871, Japan

3. Center for Immunology, University of Texas Southwestern Medical Center, Dallas, TX 75390

Abstract

Tetraspanins CD9 and CD81 facilitate the fusion between gametes, myoblasts, or virus-infected cells. Here, we investigated the role of these tetraspanins in the fusion of mononuclear phagocytes. Expression of CD9 and CD81 and their complex formation with integrins were up-regulated when blood monocytes were cultured under normal conditions. Under fusogenic conditions in the presence of Con A, CD9 and CD81 up-regulation was inhibited, and their complex formation with integrins was down-regulated. Anti-CD9 and -CD81 antibodies, which were previously shown to inhibit the fusion of gametes, myoblasts, and virus-infected cells, unexpectedly promoted the fusion of monocytes and alveolar macrophages. However, these effects were not due to altered cell adhesion, aggregation, or cytokine production. When stimulated in vitro or in vivo, alveolar macrophages and bone marrow cells of CD9- and CD81-null mice formed larger numbers of multinucleated cells than those of wild-type mice. Finally, CD9/CD81 double-null mice spontaneously developed multinucleated giant cells in the lung and showed enhanced osteoclastogenesis in the bone. These results suggest that CD9 and CD81 coordinately prevent the fusion of mononuclear phagocytes.

Publisher

Rockefeller University Press

Subject

Cell Biology

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