Bone Density Ligand, Sclerostin, Directly Interacts With LRP5 but Not LRP5G171V to Modulate Wnt Activity-Reference-Cited by-同舟云学术

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Bone Density Ligand, Sclerostin, Directly Interacts With LRP5 but Not LRP5G171V to Modulate Wnt Activity

Published:2006-08-21 Issue:11 Volume:21 Page:1738-1749
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Ellies Debra L,Viviano Beth,McCarthy John,Rey Jean-Philippe,Itasaki Nobue,Saunders Scott,Krumlauf Robb

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Reference42 articles.

1. Increased bone density in sclerosteosis is due to the deficiency of a novel secreted protein (SOST);Balemans;Hum Mol Genet,2001

2. Sclerostin is an osteocyte-expressed negative regulator of bone formation, but not a classical BMP antagonist;van Bezooijen;J Exp Med,2004

3. SOST is a ligand for LRP5/LRP6 and a WNT signaling inhibitor;Semenov;J Biol Chem,2005

4. Wise, a context-dependent activator and inhibitor of Wnt signalling;Itasaki;Development,2003

5. A mutation in the LDL receptor-related protein 5 gene results in the autosomal dominant high-bone-mass trait;Little;Am J Hum Genet,2002

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