Linked Deficiencies in Extracellular PPi and Osteopontin Mediate Pathologic Calcification Associated With Defective PC-1 and ANK Expression-Reference-Cited by-同舟云学术

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Linked Deficiencies in Extracellular PPi and Osteopontin Mediate Pathologic Calcification Associated With Defective PC-1 and ANK Expression

Published:2003-06-01 Issue:6 Volume:18 Page:994-1004
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Johnson Kristen,Goding James,Van Etten Deborah,Sali Adnan,Hu Shou-Ih,Farley David,Krug Hollis,Hessle Lovisa,Millán José Luis,Terkeltaub Robert

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Reference44 articles.

1. Matrix vesicle plasma cell membrane glycoprotein-1 regulates mineralization by murine osteoblastic MC3T3 cells;Johnson;J Bone Miner Res,1999

2. Differential mechanisms of PPi production by plasma cell membrane glycoprotein-1 (PC-1) and B10 in chondrocytes;Johnson;Arthritis Rheum,1999

3. Expression of the nucleoside triphosphate pyrophosphohydrolase PC-1 is induced by basic fibroblast growth factor (bFGF) and modulated by activation of the protein kinase A and C pathways in osteoblast-like osteosarcoma cells;Solan;J Bone Miner Res,1996

4. Osteoblast tissue-nonspecific alkaline phosphatase antagonizes and regulates PC-1;Johnson;Am J Physiol Cell Physiol,2000

5. Ectophosphodiesterase pyrophosphatase of lymphocytes and nonlymphoid cells: Structure and function of the PC-1 family;Goding;Immunol Rev,1998

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