Oral Administration of Gintonin Attenuates Cholinergic Impairments by Scopolamine, Amyloid-β Protein, and Mouse Model of Alzheimer’s Disease-Reference-Cited by-同舟云学术

Oral Administration of Gintonin Attenuates Cholinergic Impairments by Scopolamine, Amyloid-β Protein, and Mouse Model of Alzheimer’s Disease

Published:2015-08-07 Issue:9 Volume:38 Page:796-805
ISSN:1016-8478
Container-title:Molecules and Cells
language:en
Short-container-title:

Author:

Kim Hyeon-Joong,Shin Eun-Joo,Lee Byung-Hwan,Choi Sun-Hye,Jung Seok-Won,Cho Ik-Hyun,Hwang Sung-Hee,Kim Joon Yong,Han Jung-Soo,Chung ChiHye,Jang Choon-Gon,Rhim Hyewon,Kim Hyoung-Chun,Nah Seung-Yeol

Publisher

Korean Society for Molecular and Cellular Biology

Subject

Cell Biology,Molecular Biology,General Medicine

Link

http://ocean.kisti.re.kr/downfile/crosscheck/ksmcb/JAKO201530856746524.pdf

Reference46 articles.

1. Cholinergic dysfunction in a mouse model of Alzheimer disease is reversed by an anti-A antibody

2. The Cholinergic Hypothesis of Geriatric Memory Dysfunction

3. Occurrence of neuropil threads in the senile human brain and in Alzheimer's disease: A third location of paired helical filaments outside of neurofibrillary tangles and neuritic plaques

4. Exploratory, anxiety and spatial memory impairments are dissociated in mice lacking the LPA1 receptor

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