Fatsioside A-induced apoptotic death of HepG2 cells requires activation of AMP-activated protein kinase
Author:
Publisher
Spandidos Publications
Subject
Cancer Research,Oncology,Genetics,Molecular Biology,Molecular Medicine,Biochemistry
Cited by 6 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. MiR-3682 promotes the progression of hepatocellular carcinoma (HCC) via inactivating AMPK signaling by targeting ADRA1A;Annals of Hepatology;2022-01
2. 4-Acetylantrocamol LT3, a New Ubiquinone from Antrodia cinnamomea, Inhibits Hepatocellular Carcinoma HepG2 Cell Growth by Targeting YAP/TAZ, mTOR, and WNT/β-Catenin Signaling;The American Journal of Chinese Medicine;2020-01
3. The Role of AMP-Activated Protein Kinase as a Potential Target of Treatment of Hepatocellular Carcinoma;Cancers;2019-05-10
4. Integrated multi‑omics data analysis identifying novel drug sensitivity‑associated molecular targets of hepatocellular carcinoma cells;Oncology Letters;2018-05-04
5. Progression of conventional hepatic cell culture models to bioengineered HepG2 cells for evaluation of herbal bioactivities;Biotechnology Letters;2018-04-03
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