Topical Application of S1P2 Antagonist JTE-013 Attenuates 2,4-Dinitrochlorobenzene-Induced Atopic Dermatitis in Mice-Reference-Cited by-同舟云学术

Topical Application of S1P2 Antagonist JTE-013 Attenuates 2,4-Dinitrochlorobenzene-Induced Atopic Dermatitis in Mice

Published:2020-11-01 Issue:6 Volume:28 Page:537-541
ISSN:1976-9148
Container-title:Biomolecules & Therapeutics
language:en
Short-container-title:Biomolecules & Therapeutics

Author:

Kang Jisoo¹,Lee Ju-Hyun²,Im Dong-Soon¹²

Affiliation:

1. Laboratory of Pharmacology, College of Pharmacy, Pusan National University, Busan 46241, Republic of Korea

2. Laboratory of Pharmacology, College of Pharmacy, and Department of Life and Nanopharmaceutical Sciences, Graduate School, Kyung Hee University, Seoul 02447, Republic of Korea

Funder

National Research Foundation of Korea

Ministry of Education, Science and Technology

Publisher

The Korean Society of Applied Pharmacology

Subject

Drug Discovery,Pharmacology,Molecular Medicine,Biochemistry

Link

http://pdf.medrang.co.kr/BT2/2020/028/BT028-06-06.pdf

Reference26 articles.

1. Decreased Concentration and Enhanced Metabolism of Sphingosine-1-Phosphate in Lesional Skin of Dogs with Atopic Dermatitis: Disturbed Sphingosine-1-Phosphate Homeostasis in Atopic Dermatitis

2. Sphingosine 1-phospate differentially modulates maturation and function of human Langerhans-like cells

3. Circulating levels of sphingosine-1-phosphate are elevated in severe, but not mild psoriasis and are unresponsive to anti-TNF-α treatment

4. Sphingosine 1-Phosphate Modulates Antigen Capture by Murine Langerhans Cells via the S1P2 Receptor Subtype

5. Sphingosine kinase 1 activation enhances epidermal innate immunity through sphingosine-1-phosphate stimulation of cathelicidin production

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