Lipid Synthesis Is Required to Resolve Endoplasmic Reticulum Stress and Limit Fibrotic Responses in the Lung-Reference-Cited by-同舟云学术

Lipid Synthesis Is Required to Resolve Endoplasmic Reticulum Stress and Limit Fibrotic Responses in the Lung

Published:2018-08 Issue:2 Volume:59 Page:225-236
ISSN:1044-1549
Container-title:American Journal of Respiratory Cell and Molecular Biology
language:en
Short-container-title:Am J Respir Cell Mol Biol

Author:

Romero Freddy¹²,Hong Xu¹²³,Shah Dilip¹²,Kallen Caleb B.⁴,Rosas Ivan⁵,Guo Zhi⁶,Schriner DeLeila¹²,Barta Julie¹²,Shaghaghi Hoora¹²,Hoek Jan B.⁷,Mesaros Clementina⁸⁹,Choi Augustine M.¹⁰,Snyder Nathaniel W.¹¹,Summer Ross¹²

Affiliation:

1. Center for Translational Medicine

2. Jane and Leonard Korman Lung Center

3. Medical Research Center, North China University of Science and Technology, Tangshan, China

4. Department of Obstetrics and Gynecology, and

5. Division of Pulmonary and Critical Care Medicine, Department of Medicine, Brigham and Women’s Hospital, Boston, Massachusetts

6. Department of Pulmonary Diseases, Jinan Military General Hospital, Jinan, China

7. Department of Pathology, Anatomy, and Cell Biology, Thomas Jefferson University, Philadelphia, Pennsylvania

8. University of Pennsylvania Superfund Research and Training Program (Penn SRP) Center and Center of Excellence in Environmental Toxicology, and

9. Department of Systems Pharmacology and Translational Therapeutics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania

10. Joan and Sanford I. Weill Department of Medicine, New York-Presbyterian Hospital, Weill Cornell Medical College, New York, New York; and

11. A. J. Drexel Autism Institute, Drexel University, Philadelphia, Pennsylvania

Publisher

American Thoracic Society

Subject

Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Link

https://www.atsjournals.org/doi/pdf/10.1165/rcmb.2017-0340OC

Reference23 articles.

1. ER Stress Triggers Apoptosis by Activating BH3-Only Protein Bim

2. The Unfolded Protein Response: From Stress Pathway to Homeostatic Regulation

3. Lipid disequilibrium disrupts ER proteostasis by impairing ERAD substrate glycan trimming and dislocation

4. Endoplasmic Reticulum Stress Links Obesity, Insulin Action, and Type 2 Diabetes

5. Chemical Chaperones Reduce ER Stress and Restore Glucose Homeostasis in a Mouse Model of Type 2 Diabetes

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