Rhinovirus Infection Induces Interleukin-13 Production from CD11b-Positive, M2-Polarized Exudative Macrophages-Reference-Cited by-同舟云学术

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Rhinovirus Infection Induces Interleukin-13 Production from CD11b-Positive, M2-Polarized Exudative Macrophages

Published:2015-02 Issue:2 Volume:52 Page:205-216
ISSN:1044-1549
Container-title:American Journal of Respiratory Cell and Molecular Biology
language:en
Short-container-title:Am J Respir Cell Mol Biol

Author:

Chung Yutein,Hong Jun Young,Lei Jing,Chen Qiang,Bentley J. Kelley,Hershenson Marc B.

Publisher

American Thoracic Society

Subject

Cell Biology,Clinical Biochemistry,Pulmonary and Respiratory Medicine,Molecular Biology

Reference43 articles.

1. Rhinovirus Infection of Allergen-Sensitized and -Challenged Mice Induces Eotaxin Release from Functionally Polarized Macrophages

2. Rhinovirus colocalizes with CD68- and CD11b-positive macrophages following experimental infection in humans

3. Requirement for IL-13 Independently of IL-4 in Experimental Asthma

4. Pulmonary expression of interleukin-13 causes inflammation, mucus hypersecretion, subepithelial fibrosis, physiologic abnormalities, and eotaxin production

5. Persistent activation of an innate immune response translates respiratory viral infection into chronic lung disease

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3. Remember the Airway Smooth Muscle! How Rhinovirus Impairs Bronchodilator Responses;American Journal of Respiratory Cell and Molecular Biology;2023-08

4. M2 Macrophages Upregulated by Allergen Exposure in Seasonal Allergic Rhinitis;International Archives of Allergy and Immunology;2023

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