Transgenic Mice and Modeling Alzheimer's Disease
Author:
Publisher
Walter de Gruyter GmbH
Subject
General Neuroscience
Link
https://www.degruyter.com/document/doi/10.1515/REVNEURO.1995.6.2.87/pdf
Reference84 articles.
1. EARLY SENILE PLAQUES IN DOWN'S SYNDROME BRAINS SHOW A CLOSE RELATIONSHIP WITH CELL BODIES OF NEURONS
2. Neurobiology of Alzheimer's Disease
3. Attenuated protein kinase C activity and translocation in Alzheimer's Disease brain
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1. Swedish mutant APP-based BACE1 binding site peptide reduces APP β-cleavage and cerebral Aβ levels in Alzheimer’s mice;Scientific Reports;2015-06-19
2. Alzheimer's disease: Old problem, new views from transgenic and viral models;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2010-10
3. Relative Increase in Alzheimer's Disease of Soluble Forms of Cerebral Aβ Amyloid Protein Precursor Containing the Kunitz Protease Inhibitory Domain;Journal of Biological Chemistry;1998-02
4. Chapter 2. Alzheimer's Disease: Recent Advances on the Amyloid Hypothesis;Annual Reports in Medicinal Chemistry;1997
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