Swedish mutant APP-based BACE1 binding site peptide reduces APP β-cleavage and cerebral Aβ levels in Alzheimer’s mice-Reference-Cited by-同舟云学术

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Swedish mutant APP-based BACE1 binding site peptide reduces APP β-cleavage and cerebral Aβ levels in Alzheimer’s mice

Published:2015-06-19 Issue:1 Volume:5 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Li Song,Hou Huayan,Mori Takashi,Sawmiller Darrell,Smith Adam,Tian Jun,Wang Yanjiang,Giunta Brian,Sanberg Paul R.,Zhang Sheqing,Tan Jun

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep11322.pdf

Reference51 articles.

1. Selkoe, D. J. Translating cell biology into therapeutic advances in Alzheimer’s disease. Nature 399, A23–A31 (1999).

2. Sinha, S. & Lieberburg, I. Cellular mechanisms of β-amyloid production and secretion. Proc Natl Acad Sci U S A 96, 11049–11053 (1999).

3. Games, D. et al. Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein. Nature 373, 523–527 (1995).

4. Higgins, L. S. & Cordell, B. Transgenic mice and modeling Alzheimer’s disease. Rev Neurosci 6, 87–96 (1995).

5. Seiffert, D. et al. Presenilin-1 and -2 are molecular targets for γ-secretase inhibitors. J Biol Chem 275, 34086–34091 (2000).

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