The stress kinase GCN2 does not mediate suppression of antitumor T cell responses by tryptophan catabolism in experimental melanomas-Reference-Cited by-同舟云学术

The stress kinase GCN2 does not mediate suppression of antitumor T cell responses by tryptophan catabolism in experimental melanomas

Published:2016-11-15 Issue:12 Volume:5 Page:e1240858
ISSN:2162-402X
Container-title:OncoImmunology
language:en
Short-container-title:OncoImmunology

Author:

Sonner Jana K.,Deumelandt Katrin,Ott Martina,Thomé Carina M.,Rauschenbach Katharina J.,Schulz Sandra,Munteanu Bogdan,Mohapatra Soumya,Adam Isabell,Hofer Ann-Cathrin,Feuerer Markus,Opitz Christiane A.,Hopf Carsten,Wick Wolfgang,Platten Michael

Publisher

Informa UK Limited

Subject

Oncology,Immunology,Immunology and Allergy

Link

https://www.tandfonline.com/doi/pdf/10.1080/2162402X.2016.1240858

Reference41 articles.

1. IDO inhibitors move center stage in immuno-oncology

2. Evidence for a tumoral immune resistance mechanism based on tryptophan degradation by indoleamine 2,3-dioxygenase

3. An endogenous tumour-promoting ligand of the human aryl hydrocarbon receptor

4. Reversal of tumoral immune resistance by inhibition of tryptophan 2,3-dioxygenase

5. An Interaction between Kynurenine and the Aryl Hydrocarbon Receptor Can Generate Regulatory T Cells

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