The Immunomodulatory Activity of C3 Binding Glycoprotein (C3bgp) is Mediated by the Complement Receptor Type III and Mitogen-Activated Protein Kinase Signal Transduction Pathways-Reference-Cited by-同舟云学术

The Immunomodulatory Activity of C3 Binding Glycoprotein (C3bgp) is Mediated by the Complement Receptor Type III and Mitogen-Activated Protein Kinase Signal Transduction Pathways

Published:2007-01 Issue:3-4 Volume:29 Page:549-562
ISSN:0892-3973
Container-title:Immunopharmacology and Immunotoxicology
language:en
Short-container-title:Immunopharmacology and Immunotoxicology

Author:

Dobreva Zlatka Georgieva,Stanilova Spaska Angelova

Publisher

Informa UK Limited

Subject

Pharmacology,Toxicology,General Medicine,Immunology,Immunology and Allergy

Link

http://www.tandfonline.com/doi/pdf/10.1080/08923970701691017

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1. Lipopolysaccharide Induction of the Tumor Necrosis Factor-α Promoter in Human Monocytic Cells

2. Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) is required for lipopolysaccharide stimulation of tumor necrosis factor alpha (TNF-alpha) translation: glucocorticoids inhibit TNF-alpha translation by blocking JNK/SAPK

3. Endotoxin signal transduction in macrophages

4. Lipopolysaccharide-Induced Tumor Necrosis Factor Alpha Production by Human Monocytes Involves the Raf-1/MEK1-MEK2/ERK1-ERK2 Pathway

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