Use of X-chromosome inactivation pattern and laser microdissection to determine the clonal origin of focal nodular hyperplasia of the liver

Author:

Gong Li,Li Yan-Hong,Su Qin,Li Gang,Zhang Wen-Dong,Zhang Wei

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference45 articles.

1. Focal nodular hyperplasia of the liver: a comprehensive pathologic study of 305 lesions and recognition of a new histologic form;Nguyen;Am J Surg Pathol,1999

2. Clonality analysis technology based on X chromosome genetic polymorphism and application;Su;Zhonghua Bing Li Xue Za Zhi,2002

3. Use of restriction fragment length polymorphism to determine the clonal origin of human tumors;Vogelstein;Science,1985

4. Methylation of Hpa II and Hha I sites near the polymorphic CAG repeat in the human androgen-receptor gene correlates with X-chromosome inactivation;Allen;Am J Hum Genet,1992

5. Mammalian X-chromosome inactivation;Gartler;Annu Rev Genet,1983

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2. Multiclonal tumor origin: Evidence and implications;Mutation Research/Reviews in Mutation Research;2018-07

3. Focal Nodular Hyperplasia (FNH) of the Liver;Tumors and Tumor-Like Lesions of the Hepatobiliary Tract;2016-10-20

4. Clinical Features of Focal Nodular Hyperplasia of the Liver in Children;Journal of Pediatric Gastroenterology & Nutrition;2016-06

5. Focal Nodular Hyperplasia (FNH) of the Liver;Tumors and Tumor-Like Lesions of the Hepatobiliary Tract;2016

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