The structure of CLEC-2: mechanisms of dimerization and higher-order clustering-Reference-Cited by-同舟云学术

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The structure of CLEC-2: mechanisms of dimerization and higher-order clustering

Published:2021-04-05 Issue:6 Volume:32 Page:733-743
ISSN:0953-7104
Container-title:Platelets
language:en
Short-container-title:Platelets

Author:

Martin Eleyna M¹^ORCID,Zuidscherwoude Malou¹^ORCID,Morán Luis a¹²^ORCID,Di Ying¹,García Angel²,Watson Steve P¹³^ORCID

Affiliation:

1. Institute of Cardiovascular Sciences, College of Medical and Dental Sciences, University of Birmingham, Edgbaston, Birmingham

2. Centre for Research in Molecular Medicine and Chronic Diseases (CIMUS), Universidade De Santiago De Compostela, Spain

3. Centre of Membrane Proteins and Receptors (COMPARE), Universities of Birmingham and Nottingham, The Midlands

Funder

British Heart Foundation

Marie Sklodowska-Curie

Wellcome Trust

Publisher

Informa UK Limited

Subject

Hematology,General Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/09537104.2021.1906407

Reference69 articles.

1. Molecular characterization of two novel C-type lectin-like receptors, one of which is selectively expressed in human dendritic cells

2. Functional significance of the platelet immune receptors GPVI and CLEC-2

3. A novel Syk-dependent mechanism of platelet activation by the C-type lectin receptor CLEC-2

4. Renal cells activate the platelet receptor CLEC-2 through podoplanin

Cited by 26 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The podoplanin-CLEC-2 interaction promotes platelet-mediated melanoma pulmonary metastasis;BMC Cancer;2024-04-01

2. C-type lectin-like receptor 2: roles and drug target;Thrombosis Journal;2024-03-19

3. A role of platelet C-type lectin-like receptor-2 and its ligand podoplanin in vascular biology;Current Opinion in Hematology;2024-02-06

4. A change of rhodocytin's suprastructure turns the agonist into an antagonist of tumor cell induced platelet aggregation;The FASEB Journal;2024-01-09

5. Trivalent nanobody-based ligands mediate powerful activation of GPVI, CLEC-2, and PEAR1 in human platelets whereas FcγRIIA requires a tetravalent ligand;Journal of Thrombosis and Haemostasis;2024-01

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