Enzyme replacement therapy with recombinant pro-CTSD (cathepsin D) corrects defective proteolysis and autophagy in neuronal ceroid lipofuscinosis-Reference-Cited by-同舟云学术

Enzyme replacement therapy with recombinant pro-CTSD (cathepsin D) corrects defective proteolysis and autophagy in neuronal ceroid lipofuscinosis

Published:2019-07-16 Issue:5 Volume:16 Page:811-825
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Marques André R. A.¹^ORCID,Di Spiezio Alessandro¹^ORCID,Thießen Niklas¹,Schmidt Lina¹,Grötzinger Joachim¹,Lüllmann-Rauch Renate²,Damme Markus¹,Storck Steffen E.³^ORCID,Pietrzik Claus U.³,Fogh Jens⁴,Bär Julia⁵,Mikhaylova Marina⁵^ORCID,Glatzel Markus⁶^ORCID,Bassal Mahmoud⁷^ORCID,Bartsch Udo⁷,Saftig Paul¹^ORCID

Affiliation:

1. Institute of Biochemistry, Christian-Albrechts-University Kiel, Kiel, Germany

2. Institute of Anatomy, Christian-Albrechts-University Kiel, Kiel, Germany

3. Institute for Pathobiochemistry, University Medical Center of Johannes Gutenberg University Mainz, Mainz, Germany

4. OrfoNeuro ApS, Lynge, Denmark

5. Center for Molecular Neurobiology, Emmy-Noether Group “Neuronal Protein Transport”, ZMNH, University Medical Center, Hamburg, Germany

6. Institute of Neuropathology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany

7. Department of Ophthalmology, Experimental Ophthalmology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany

Funder

Bundesministerium für Forschung und Technologie

Deutsche Forschungsgemeinschaft

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2019.1637200

Reference38 articles.

1. Participation of Autophagy in Storage of Lysosomes in Neurons from Mouse Models of Neuronal Ceroid-Lipofuscinoses (Batten Disease)

2. The Role of Cathepsin D in the Pathogenesis of Human Neurodegenerative Disorders

3. Lrp1/LDL Receptor Play Critical Roles in Mannose 6-Phosphate-Independent Lysosomal Enzyme Targeting

4. Processing of Human Cathepsin D Is Independent of Its Catalytic Function and Auto-Activation: Involvement of Cathepsins L and B

5. Cathepsin D Deficiency Is Associated with a Human Neurodegenerative Disorder

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