Unravelling the complex interplay of cuproptosis, lncRNAs, and immune infiltration in Alzheimer’s disease: a step towards novel therapeutic targets-Reference-Cited by-同舟云学术

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Unravelling the complex interplay of cuproptosis, lncRNAs, and immune infiltration in Alzheimer’s disease: a step towards novel therapeutic targets

Published:2024-05-21 Issue:1 Volume:51 Page:
ISSN:0301-4460
Container-title:Annals of Human Biology
language:en
Short-container-title:Annals of Human Biology

Author:

Zeng Yi¹,Qian Siqi¹,Cao Yuan¹,Xiao Wenbiao¹

Affiliation:

1. Department of Geriatrics, the Second Xiangya Hospital of Central South University, Changsha, Hunan, China

Funder

National Science & Technology Fundamental Resources Investigation Program of China

the National Natural Science Foundation of China

the Hunan Provincial Natural Science Foundation of China

Publisher

Informa UK Limited

Link

https://www.tandfonline.com/doi/pdf/10.1080/03014460.2024.2342531

Reference54 articles.

1. Suppression of a core metabolic enzyme dihydrolipoamide dehydrogenase (dld) protects against amyloid beta toxicity in C. elegans model of Alzheimer's disease

2. Dihydrolipoamide dehydrogenase suppression induces human tau phosphorylation by increasing whole body glucose levels in a C. elegans model of Alzheimer’s Disease

3. Gene Expression Biomarkers in the Brain of a Mouse Model for Alzheimer's Disease: Mining of Microarray Data by Logic Classification and Feature Selection

4. The Perspective of Dysregulated LncRNAs in Alzheimer's Disease: A Systematic Scoping Review

5. Characterization of Copper Interactions with Alzheimer Amyloid β Peptides

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