Dihydrolipoamide dehydrogenase suppression induces human tau phosphorylation by increasing whole body glucose levels in a C. elegans model of Alzheimer’s Disease-Reference-Cited by-同舟云学术

Dihydrolipoamide dehydrogenase suppression induces human tau phosphorylation by increasing whole body glucose levels in a C. elegans model of Alzheimer’s Disease

Published:2018-07-28 Issue:11 Volume:236 Page:2857-2866
ISSN:0014-4819
Container-title:Experimental Brain Research
language:en
Short-container-title:Exp Brain Res

Author:

Ahmad Waqar^ORCID

Publisher

Springer Science and Business Media LLC

Subject

General Neuroscience

Link

http://link.springer.com/article/10.1007/s00221-018-5341-0/fulltext.html

Reference65 articles.

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2. Ahmad W (2013) Overlapped metabolic and therapeutic links between Alzheimer and diabetes. Mol Neurobiol 47(1):399–424. https://doi.org/10.1007/s12035-012-8352-z

3. Ahmad W, Ebert PR (2016) Metformin attenuates abeta pathology mediated through levamisole sensitive nicotinic acetylcholine receptors in a C. elegans model of Alzheimer’s disease. Mol Neurobiol 54(7):5427–5439. https://doi.org/10.1007/s12035-016-0085-y

4. Ahmad W, Ebert PR (2018a) 5-Methoxyindole-2-carboxylic acid (MICA) suppresses Abeta-mediated pathology in C. elegans. Exp Gerontol 108:215–225. https://doi.org/10.1016/j.exger.2018.04.021

5. Ahmad W, Ebert PR (2018b) Suppression of the dihydrolipoamide dehydrogenase gene (dld-1) protects against the toxicity of human amyloid beta in C. elegans model of Alzheimer’s disease. bioRxiv 228429

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