N6-methyladenosine-dependent primary microRNA-126 processing activated PI3K-AKT-mTOR pathway drove the development of pulmonary fibrosis induced by nanoscale carbon black particles in rats-Reference-Cited by-同舟云学术

N6-methyladenosine-dependent primary microRNA-126 processing activated PI3K-AKT-mTOR pathway drove the development of pulmonary fibrosis induced by nanoscale carbon black particles in rats

Published:2019-09-10 Issue:1 Volume:14 Page:1-20
ISSN:1743-5390
Container-title:Nanotoxicology
language:en
Short-container-title:Nanotoxicology

Author:

Han Bin¹,Chu Chen¹,Su Xuan¹,Zhang Ning¹,Zhou Lixiao¹,Zhang Mengyue¹,Yang Shuaishuai¹,Shi Lei²,Zhao Bo³,Niu Yujie²⁴,Zhang Rong¹⁴

Affiliation:

1. Department of Toxicology, School of Public Health, Hebei Medical University, Shijiazhuang, China;

2. Occupational Health and Environmental Health, School of Public Health, Hebei Medical University, Shijiazhuang, China;

3. Department of Laboratory Diagnosis, Hebei Medical University, Shijiazhuang, China;

4. Hebei Key Laboratory of Environment and Human Health, Hebei Medical University, Shijiazhuang, China

Publisher

Informa UK Limited

Subject

Toxicology,Biomedical Engineering

Link

https://www.tandfonline.com/doi/pdf/10.1080/17435390.2019.1661041

Reference83 articles.

1. HNRNPA2B1 Is a Mediator of m6A-Dependent Nuclear RNA Processing Events

2. N6-methyladenosine marks primary microRNAs for processing

3. The functions of animal microRNAs

4. Inhalation of ultrafine carbon particles triggers biphasic pro-inflammatory response in the mouse lung

5. A Multiple-Path Model of Particle Deposition in the Rat Lung

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