Anti-BCMA antibodies in the future management of multiple myeloma
Author:
Affiliation:
1. Oncology Department, Department of Therapeutics, Alexandra Hospital, National and Kapodistrian University of Athens, Athens, Greece
Funder
funded
Publisher
Informa UK Limited
Subject
Pharmacology (medical),Oncology
Link
https://www.tandfonline.com/doi/pdf/10.1080/14737140.2019.1586539
Reference35 articles.
1. A new gene, BCM, on chromosome 16 is fused to the interleukin 2 gene by a t(4;16)(q26;p13) translocation in a malignant T cell lymphoma.
2. The BCMA gene, preferentially expressed during B lymphoid maturation, is bidirectionally transcribed
3. Expression of BCMA, TACI, and BAFF-R in multiple myeloma: a mechanism for growth and survival
4. Baff Binds to the Tumor Necrosis Factor Receptor–Like Molecule B Cell Maturation Antigen and Is Important for Maintaining the Peripheral B Cell Population
5. Target Expression, Generation, Preclinical Activity, and Pharmacokinetics of the BCMA-T Cell Bispecific Antibody EM801 for Multiple Myeloma Treatment
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