Exploration of the mechanism of NORAD activation of TGF-β1/Smad3 through miR-136-5p and promotion of tacrolimus-induced renal fibrosis
Author:
Affiliation:
1. Department of Kidney Transplantation, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China
2. Department of Nephrology, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China
Publisher
Informa UK Limited
Subject
Nephrology,Critical Care and Intensive Care Medicine,General Medicine
Link
https://www.tandfonline.com/doi/pdf/10.1080/0886022X.2022.2147083
Reference22 articles.
1. Optimization of tacrolimus in kidney transplantation: New pharmacokinetic perspectives
2. Tacrolimus increases the expression level of the chemokine receptor CXCR2 to promote renal fibrosis progression;Wang D;Int J Mol Med,2019
3. Tacrolimus Modulates TGF-β Signaling to Induce Epithelial-Mesenchymal Transition in Human Renal Proximal Tubule Epithelial Cells
4. miR-136 improves renal fibrosis in diabetic rats by targeting down-regulation of tyrosine kinase SYK and inhibition of TGF-β1/Smad3 signaling pathway
5. Effect of miRNA on the TGF-beta/smad signal pathway in hepatic fibrosis;Ma C;Chin J Clin Anat,2021
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