Tacrolimus Modulates TGF-β Signaling to Induce Epithelial-Mesenchymal Transition in Human Renal Proximal Tubule Epithelial Cells-Reference-Cited by-同舟云学术

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Tacrolimus Modulates TGF-β Signaling to Induce Epithelial-Mesenchymal Transition in Human Renal Proximal Tubule Epithelial Cells

Published:2016-04-26 Issue:5 Volume:5 Page:50
ISSN:2077-0383
Container-title:Journal of Clinical Medicine
language:en
Short-container-title:JCM

Author:

Bennett Jason,Cassidy Hilary^ORCID,Slattery Craig,Ryan Michael,McMorrow Tara

Publisher

MDPI AG

Subject

General Medicine

Link

http://www.mdpi.com/2077-0383/5/5/50/pdf

Reference84 articles.

1. International Comparison of the Relationship of Chronic Kidney Disease Prevalence and ESRD Risk

2. Prevalence of Chronic Kidney Disease in the United States

3. Prevalence of Chronic Kidney Disease in Persons With Undiagnosed or Prehypertension in the United States

4. Chronic kidney disease: a public health priority and harbinger of premature cardiovascular disease

5. Interaction of Aging and Chronic Kidney Disease

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1. Enhanced eNOS/nitric oxide production by nebivolol interferes with TGF-β1/Smad3 signaling and collagen I deposition in the kidney after prolonged tacrolimus administration;Life Sciences;2024-10

2. The protective effect of 1400W against ischaemia and reperfusion injury is countered by transient medullary kidney endothelial dysregulation;The Journal of Physiology;2024-07-26

3. A multi-omics investigation of tacrolimus off-target effects on a proximal tubule cell-line;Pharmacological Research;2023-06

4. Tacrolimus induces fibroblast-to-myofibroblast transition via a TGF-β-dependent mechanism to contribute to renal fibrosis;American Journal of Physiology-Renal Physiology;2023-05-01

5. Notoginsenoside R1 prevents EMT of BEAS-2B cells via suppressing the TGF-β1/Smad pathway;2023-03-01

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