A mitochondrial-targeted peptide ameliorated podocyte apoptosis through a HOCl-alb-enhanced and mitochondria-dependent signalling pathway in diabetic rats and in vitro-Reference-Cited by-同舟云学术

A mitochondrial-targeted peptide ameliorated podocyte apoptosis through a HOCl-alb-enhanced and mitochondria-dependent signalling pathway in diabetic rats and in vitro

Published:2019-01-01 Issue:1 Volume:34 Page:394-404
ISSN:1475-6366
Container-title:Journal of Enzyme Inhibition and Medicinal Chemistry
language:en
Short-container-title:Journal of Enzyme Inhibition and Medicinal Chemistry

Author:

Wang Xiaoqiao¹,Tang Dongdong²,Zou Yaowei¹,Wu Xiaoyu¹,Chen Yihua¹,Li Hongying³,Chen Siqi¹,Shi Yue³,Niu Hongxin³^ORCID

Affiliation:

1. Division of Nephrology, Zhujiang Hospital, Southern Medical University, Guangzhou, China;

2. Division of Nephrology, Huadu District people's Hospital, Southern Medical University, Guangzhou, China;

3. Special Medical Service Center, Zhujiang Hospital, Southern Medical University, Guangzhou, China

Funder

National Natural Science Foundation of China

NSFC

Science and Technology Planning Project of Guangdong Province

Publisher

Informa UK Limited

Subject

Drug Discovery,Pharmacology,General Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/14756366.2018.1488697

Reference43 articles.

1. A Systems Biology Overview on Human Diabetic Nephropathy: From Genetic Susceptibility to Post-Transcriptional and Post-Translational Modifications

2. Associations of kidney disease measures with mortality and end-stage renal disease in individuals with and without diabetes: a meta-analysis

3. Podocyte loss and progressive glomerular injury in type II diabetes.

4. Podocyte detachment and reduced glomerular capillary endothelial fenestration promote kidney disease in type 2 diabetic nephropathy

5. Glucose-Induced Reactive Oxygen Species Cause Apoptosis of Podocytes and Podocyte Depletion at the Onset of Diabetic Nephropathy

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