Substance P increases CCN2 dependent on TGF-beta yet Collagen Type I via TGF-beta1 dependent and independent pathways in tenocytes
Author:
Affiliation:
1. Department of Anatomy and Cell Biology, Lewis Katz School of Medicine, Temple University, Philadelphia, PA, USA
Funder
National Institute of Arthritis and Musculoskeletal and Skin Diseases
Publisher
Informa UK Limited
Subject
Cell Biology,Molecular Biology,Orthopedics and Sports Medicine,Biochemistry,Rheumatology
Link
https://www.tandfonline.com/doi/pdf/10.1080/03008207.2017.1297809
Reference57 articles.
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2. TGF‐β signaling and the fibrotic response
3. TGFβ signaling
4. Transforming Growth Factor 1 Improves Wound Healing and Random Flap Survival in Normal and Irradiated Rats
5. Studies in Flexor Tendon Wound Healing: Neutralizing Antibody to TGF-β1 Increases Postoperative Range of Motion
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