Lack of correlation between surface expression and currents in epileptogenic AB-calmodulin binding domain Kv7.2 potassium channel mutants
Author:
Affiliation:
1. Instituto Biofisika, Consejo Superior de Investigaciones Científicas, CSIC, UPV/EHU, Leioa, Spain
Publisher
Informa UK Limited
Subject
Biochemistry,Biophysics
Link
https://www.tandfonline.com/doi/pdf/10.1080/19336950.2018.1511512
Reference50 articles.
1. Modulation of Kv7 channels and excitability in the brain
2. Neuronal KCNQ potassium channels:physislogy and role in disease
3. Driving With No Brakes: Molecular Pathophysiology of Kv7 Potassium Channels
4. The C-terminus of Kv7 channels: a multifunctional module
5. Expression of a calmodulin-binding KCNQ2 potassium channel fragment modulates neuronal M-current and membrane excitability
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1. High-throughput evaluation of epilepsy-associated KCNQ2 variants reveals functional and pharmacological heterogeneity;JCI Insight;2022-03-08
2. High-throughput Evaluation of Epilepsy-associated KCNQ2 Variants Reveals Functional and Pharmacological Heterogeneity;2021-10-19
3. Pathogenic variants in KCNQ2 cause intellectual deficiency without epilepsy: Broadening the phenotypic spectrum of a potassium channelopathy;American Journal of Medical Genetics Part A;2021-03-23
4. The S2–S3 Loop of Kv7.4 Channels Is Essential for Calmodulin Regulation of Channel Activation;Frontiers in Physiology;2021-01-20
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