SAMHD1 protects cancer cells from various nucleoside-based antimetabolites-Reference-Cited by-同舟云学术

SAMHD1 protects cancer cells from various nucleoside-based antimetabolites

Published:2017-05-08 Issue:11 Volume:16 Page:1029-1038
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Herold Nikolas¹²^ORCID,Rudd Sean G.³^ORCID,Sanjiv Kumar³,Kutzner Juliane⁴,Bladh Julia¹^ORCID,Paulin Cynthia B. J.³^ORCID,Helleday Thomas³,Henter Jan-Inge¹²,Schaller Torsten⁴^ORCID

Affiliation:

1. Childhood Cancer Research Unit, Department of Women's and Children's Health, Karolinska Institutet, Stockholm, Sweden

2. Theme of Children′s and Women′s Health, Astrid Lindgren Children's Hospital, Karolinska University Hospital, Stockholm, Sweden

3. Science for Life Laboratory, Division of Translational Medicine and Chemical Biology, Department of Medical Biochemistry and Biophysics, Karolinska Institutet, Stockholm, Sweden

4. Department of Infectious Diseases, Virology, University Hospital Heidelberg, Heidelberg, Germany

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15384101.2017.1314407

Reference90 articles.

1. SAMHD1 is the dendritic- and myeloid-cell-specific HIV-1 restriction factor counteracted by Vpx

2. Vpx relieves inhibition of HIV-1 infection of macrophages mediated by the SAMHD1 protein

3. Erratum: Corrigendum: SAMHD1 restricts the replication of human immunodeficiency virus type 1 by depleting the intracellular pool of deoxynucleoside triphosphates

4. HIV-1 restriction factor SAMHD1 is a deoxynucleoside triphosphate triphosphohydrolase

5. The deoxynucleotide triphosphohydrolase SAMHD1 is a major regulator of DNA precursor pools in mammalian cells

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