Combined immune checkpoint blockade increases CD8+CD28+PD-1+ effector T cells and provides a therapeutic strategy for patients with neuroblastoma-Reference-Cited by-同舟云学术

Combined immune checkpoint blockade increases CD8+CD28+PD-1+ effector T cells and provides a therapeutic strategy for patients with neuroblastoma

Published:2021-01-01 Issue:1 Volume:10 Page:
ISSN:2162-402X
Container-title:OncoImmunology
language:en
Short-container-title:OncoImmunology

Author:

Shirinbak Soheila¹,Chan Randall Y.¹²³,Shahani Shilpa⁴,Muthugounder Sakunthala¹,Kennedy Rebekah¹,Hung Long T.¹,Fernandez G. Esteban¹,Hadjidaniel Michael D.¹,Moghimi Babak¹³⁵,Sheard Michael A.¹,Epstein Alan L.⁵,Fabbri Muller⁶,Shimada Hiroyuki¹⁵,Asgharzadeh Shahab¹³⁵

Affiliation:

1. Department of Pediatrics, Children's Hospital Los Angeles and the Saban Research Institute, Los Angeles, CA, USA

2. Department of Pediatrics, Los Angeles County + University of Southern California Medical Center, Los Angeles, CA, USA

3. Department of Pediatrics, Keck School of Medicine, University of Southern California, Los Angeles, CA, USA

4. Center for Cancer Research, National Cancer Institute, Bethesda, MD, USA

5. Keck School of Medicine, University of Southern California, Los Angeles, CA, USA

6. Cancer Biology Program, University of Hawaii Cancer Center, Honolulu, HI, USA

Funder

the Department of Defense

National Cancer Institute

Publisher

Informa UK Limited

Subject

Oncology,Immunology,Immunology and Allergy

Link

https://www.tandfonline.com/doi/pdf/10.1080/2162402X.2020.1838140

Reference44 articles.

1. Regulation of T Cell Receptor Signaling by Tyrosine Phosphatase SYP Association with CTLA-4

2. PD-1 immunoreceptor inhibits B cell receptor-mediated signaling by recruiting src homology 2-domain-containing tyrosine phosphatase 2 to phosphotyrosine

3. In vivo imaging reveals a tumor-associated macrophage–mediated resistance pathway in anti–PD-1 therapy

4. Interactions between PD-1 and PD-L1 promote tolerance by blocking the TCR–induced stop signal

5. Temporally Distinct PD-L1 Expression by Tumor and Host Cells Contributes to Immune Escape

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