The mechanism of sodium butyrate on the growth of mouse B16 melanoma cells by inhibiting the differentiation of M2-type macrophages and down-regulating the expressions of VEGF and TGF-β-Reference-Cited by-同舟云学术

The mechanism of sodium butyrate on the growth of mouse B16 melanoma cells by inhibiting the differentiation of M2-type macrophages and down-regulating the expressions of VEGF and TGF-β

Published:2023-04-17 Issue: Volume: Page:1-11
ISSN:0264-8725
Container-title:Biotechnology and Genetic Engineering Reviews
language:en
Short-container-title:Biotechnology and Genetic Engineering Reviews

Author:

Jia Zhenhua¹,Jin Jun²,Wang Wei¹,Wang Xiaobo¹

Affiliation:

1. Department of Medical Cosmetology, The Third Affiliated Hospital of Zhengzhou University, Zhengzhou, China

2. Plastic Surgery Department, Henan Provincial People’s Hospital, Zhengzhou, China

Funder

financial support

Publisher

Informa UK Limited

Subject

Molecular Biology,Bioengineering,Biotechnology

Link

https://www.tandfonline.com/doi/pdf/10.1080/02648725.2023.2202994

Reference13 articles.

1. Overexpression of Histone Deacetylase 1 Confers Resistance to Sodium Butyrate–Mediated Apoptosis in Melanoma Cells through a p53-Mediated Pathway

2. CLINICAL DEVELOPMENT OF HISTONE DEACETYLASE INHIBITORS AS ANTICANCER AGENTS

3. Deacetylase inhibition in malignant melanomas: impact on cell cycle regulation and survival

4. Phase Ia dose escalation study of OBP-801, a cyclic depsipeptide class I histone deacetylase inhibitor, in patients with advanced solid tumors

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