Lapatinib induces mitochondrial dysfunction to enhance oxidative stress and ferroptosis in doxorubicin-induced cardiomyocytes via inhibition of PI3K/AKT signaling pathway-Reference-Cited by-同舟云学术

Lapatinib induces mitochondrial dysfunction to enhance oxidative stress and ferroptosis in doxorubicin-induced cardiomyocytes via inhibition of PI3K/AKT signaling pathway

Published:2021-12-29 Issue:1 Volume:13 Page:48-60
ISSN:2165-5979
Container-title:Bioengineered
language:en
Short-container-title:Bioengineered

Author:

Sun Lei¹,Wang Hua¹,Xu Dan¹,Yu Shanshan¹,Zhang Lin¹,Li Xiaopeng¹

Affiliation:

1. Ultrasonic Department, The Second Affiliated Hospital of Xi’an Jiaotong University, Xi’an, China

Funder

Natural Science Foundation of Shaanxi Province

Publisher

Informa UK Limited

Subject

Applied Microbiology and Biotechnology,General Medicine,Bioengineering,Biotechnology

Link

https://www.tandfonline.com/doi/pdf/10.1080/21655979.2021.2004980

Reference43 articles.

1. Doxorubicin-Induced Cardiotoxicity: From Bioenergetic Failure and Cell Death to Cardiomyopathy

2. Polyphenols, autophagy and doxorubicin-induced cardiotoxicity

3. Protective effect of limonin against doxorubicin-induced cardiotoxicity via activating nuclear factor - like 2 and Sirtuin 2 signaling pathways

4. Oxidative stress injury in doxorubicin-induced cardiotoxicity

5. Insights into Doxorubicin-induced Cardiotoxicity: Molecular Mechanisms, Preventive Strategies, and Early Monitoring

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