CALR, JAK2 and MPL mutation status in Argentinean patients with BCR-ABL1- negative myeloproliferative neoplasms
Author:
Affiliation:
1. Cátedra de Hematología, Facultad de Ciencias Bioquímicas y Farmacéuticas, Universidad Nacional de Rosario, Guaraní 3012, Rosario, Argentina
Publisher
Informa UK Limited
Subject
Hematology,Hematology
Link
https://www.tandfonline.com/doi/pdf/10.1080/10245332.2017.1385891
Reference27 articles.
1. The 2008 revision of the World Health Organization (WHO) classification of myeloid neoplasms and acute leukemia: rationale and important changes
2. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera
3. Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis
4. A Gain-of-Function Mutation ofJAK2in Myeloproliferative Disorders
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3. Frequency of JAK2V617F, MPL and CALR driver mutations and associated clinical characteristics in a Norwegian patient cohort with myeloproliferative neoplasms;Scandinavian Journal of Clinical and Laboratory Investigation;2022-12-07
4. Calreticulin Mutations in Philadelphia Chromosome Negative Myeloproliferative Neoplasms;International Journal of Hematology and Oncology;2022-04-30
5. Molecular Screening of Exon 12 of Janus Kinase 2, Exon 9 of Calreticulin Genes in Polycythemia Vera Patients with Unmutated Janus Kinase 2 V617F;Indian Journal of Pharmaceutical Sciences;2022
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