Genetic Mechanisms of ADPKD
Author:
Publisher
Springer Singapore
Link
http://link.springer.com/content/pdf/10.1007/978-981-10-2041-4_2
Reference71 articles.
1. Anyatonwu GI, Ehrlich BE (2005) Organic cation permeation through the channel formed by polycystin-2. J Biol Chem 280(33):29488–29493. doi: 10.1074/jbc.M504359200
2. Anyatonwu GI, Estrada M, Tian X, Somlo S, Ehrlich BE (2007) Regulation of ryanodine receptor-dependent calcium signaling by polycystin-2. Proc Natl Acad Sci U S A 104(15):6454–6459. doi: 10.1073/pnas.0610324104
3. Babich V, Zeng WZ, Yeh BI, Ibraghimov-Beskrovnaya O, Cai Y, Somlo S, Huang CL (2004) The N-terminal extracellular domain is required for polycystin-1-dependent channel activity. J Biol Chem 279(24):25582–25589. doi: 10.1074/jbc.M402829200
4. Banizs B, Komlosi P, Bevensee MO, Schwiebert EM, Bell PD, Yoder BK (2007) Altered pH(i) regulation and Na(+)/HCO3(−) transporter activity in choroid plexus of cilia-defective Tg737(orpk) mutant mouse. Am J Physiol Cell Physiol 292(4):C1409–C1416. doi: 10.1152/ajpcell.00408.2006
5. Bhunia AK, Piontek K, Boletta A, Liu L, Qian F, Xu PN, Germino FJ, Germino GG (2002) PKD1 induces p21(waf1) and regulation of the cell cycle via direct activation of the JAK-STAT signaling pathway in a process requiring PKD2. Cell 109(2):157–168
Cited by 38 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Acyl-CoA thioesterase 13 (ACOT13) attenuates the progression of autosomal dominant polycystic kidney disease in vitro via triggering mitochondrial-related cell apoptosis;Aging;2024-08-21
2. Investigational agents for autosomal dominant polycystic kidney disease: preclinical and early phase study insights;Expert Opinion on Investigational Drugs;2024-04-21
3. Autosomal Dominant Polycystic Kidney Disease: Extrarenal Involvement;International Journal of Molecular Sciences;2024-02-22
4. Quantifying variant contributions in cystic kidney disease using national-scale whole genome sequencing;2024-02-15
5. The Pathophysiology of Inherited Renal Cystic Diseases;Genes;2024-01-11
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3