A more detailed description of study methods could be needed: Comments on “Clinico-serological associations of urinary activated leukocyte cell adhesion molecule in systemic lupus erythematosus and lupus nephritis”
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Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s10067-024-06944-1.pdf
Reference6 articles.
1. Amer AS, Abdel Moneam SM, Hashaad NI, Yousef EM, Abd El-Hassib DM (2024) Clinico-serological associations of urinary activated leukocyte cell adhesion molecule in systemic lupus erythematosus and lupus nephritis. Clin Rheumatol 43(3):1015–1021
2. Parodis I, Gokaraju S, Zickert A, Vanarsa K et al (2020) ALCAM and VCAM-1 as urine biomarkers of activity and long-term renal outcome in systemic lupus erythematosus. Rheumatology (Oxford) 59(9):2237–2249
3. Stanley S, Vanarsa K, Soliman S, Habazi D et al (2020) Comprehensive aptamer-based screening identifies a spectrum of urinary biomarkers of lupus nephritis across ethnicities. Nat Commun 11(1):2197
4. Ding H, Lin C, Cai J, Guo Q et al (2020) Urinary activated leukocyte cell adhesion molecule as a novel biomarker of lupus nephritis histology. Arthritis Res Ther 22(1):122
5. Chalmers SA, Ayilam Ramachandran R, Garcia SJ, Der E et al (2022) The CD6/ALCAM pathway promotes lupus nephritis via T cell-mediated responses. J Clin Invest 132(1):e147334
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