Nanomolar concentrations of cocaine enhance D2-like agonist-induced inhibition of the K+-evoked [3H]-dopamine efflux from rat striatal synaptosomes: a novel action of cocaine-Reference-Cited by-同舟云学术

Nanomolar concentrations of cocaine enhance D2-like agonist-induced inhibition of the K+-evoked [3H]-dopamine efflux from rat striatal synaptosomes: a novel action of cocaine

Published:2010-03-31 Issue:5 Volume:117 Page:593-597
ISSN:0300-9564
Container-title:Journal of Neural Transmission
language:en
Short-container-title:J Neural Transm

Author:

Ferraro Luca,Beggiato Sarah,Marcellino Daniel,Frankowska Malgorzata,Filip Malgorzata,Agnati Luigi F.,Antonelli Tiziana,Tomasini Maria C.,Tanganelli Sergio,Fuxe Kjell

Publisher

Springer Science and Business Media LLC

Subject

Biological Psychiatry,Psychiatry and Mental health,Neurology (clinical),Neurology

Link

http://link.springer.com/content/pdf/10.1007/s00702-010-0389-4.pdf

Reference14 articles.

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2. Biegon A et al (1992) Quantitative autoradiography of cocaine binding sites in human brain postmortem. Synapse 10:126–130

3. Bonnet JJ, Costentin J (1986) GBR 12783, a potent and selective inhibitor of dopamine uptake: biochemical studies in vivo and ex vivo. Eur J Pharmacol 121:199–209

4. Di Chiara G, Imperato A (1988) Drugs abused by humans preferentially increase synaptic dopamine concentrations in the mesolimbic system of freely moving rats. Proc Natl Acad Sci USA 85:5274–5278

5. Fuxe K et al (2007) From the Golgi-Cajal mapping to the transmitter-based characterization of the neuronal networks leading to two modes of brain communication: wiring and volume transmission. Brain Res Rev 55:17–54

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