LncRNA SNHG16 regulates RAS and NF-κB pathway-mediated NLRP3 inflammasome activation to aggravate diabetes nephropathy through stabilizing TLR4
Author:
Funder
Guangdong Provincial Basic and Applied Basic Research Fund Regional Joint Fund Project
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology,General Medicine,Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00592-022-02021-8.pdf
Reference36 articles.
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2. Cai F, Jiang H, Li Y, Li Q, Yang C (2021) Upregulation of long non-coding RNA SNHG16 promotes diabetes-related RMEC dysfunction via activating NF-κB and PI3K/AKT pathways. Mol Ther Nucleic Acids 24:512–527. https://doi.org/10.1016/j.omtn.2021.01.035
3. D’Addio F, Vergani A, Potena L et al (2018) P2X7R mutation disrupts the NLRP3-mediated Th program and predicts poor cardiac allograft outcomes. J Clin Invest 128(8):3490–3503. https://doi.org/10.1172/jci94524
4. Duan YR, Chen BP, Chen F et al (2021) LncRNA lnc-ISG20 promotes renal fibrosis in diabetic nephropathy by inducing AKT phosphorylation through miR-486-5p/NFAT5. J Cell Mol Med 25(11):4922–4937. https://doi.org/10.1111/jcmm.16280
5. Feng Q, Liu D, Lu Y, Liu Z (2020) The interplay of renin-angiotensin system and Toll-like receptor 4 in the inflammation of diabetic nephropathy. J Immunol Res 2020:6193407. https://doi.org/10.1155/2020/6193407
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