The impact of TOPBP1 mutations in human cancers on the DNA damage response
Author:
Funder
The National Natural Science Foundation of China
Publisher
Springer Science and Business Media LLC
Subject
General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s42764-022-00072-3.pdf
Reference51 articles.
1. Adam, S., Rossi, S. E., Moatti, N., Zompit, M. D., Xue, Y. B., Ng, T. F., Durocher, D., et al. (2021). The CIP2A-TOPBP1 axis safeguards chromosome stability and is a synthetic lethal target for BRCA-mutated cancer. Nature Cancer. https://doi.org/10.1038/s43018-021-00266-w
2. Ashwell, S., Janetka, J. W., & Zabludoff, S. (2008). Keeping checkpoint kinases in line: New selective inhibitors in clinical trials. Expert Opinion on Investigational Drugs, 17(9), 1331–1340. https://doi.org/10.1517/13543784.17.9.1331
3. Blackford, A. N., Nieminuszczy, J., Schwab, R. A., Galanty, Y., Jackson, S. P., & Niedzwiedz, W. (2015). TopBP1 interacts with BLM to maintain genome stability but is dispensable for preventing BLM degradation. Molecular Cell, 57(6), 1133–1141. https://doi.org/10.1016/j.molcel.2015.02.012
4. Burrows, A. E., & Elledge, S. J. (2008). How ATR turns on: TopBP1 goes on ATRIP with ATR. Genes & Development, 22(11), 1416–1421. https://doi.org/10.1101/gad.1685108
5. Cescutti, R., Negrini, S., Kohzaki, M., & Halazonetis, T. D. (2010). TopBP1 functions with 53BP1 in the G1 DNA damage checkpoint. Embo Journal, 29(21), 3723–3732. https://doi.org/10.1038/emboj.2010.238
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