Protection of pancreatic β-cell by phosphocreatine through mitochondrial improvement via the regulation of dual AKT/IRS-1/GSK-3β and STAT3/Cyp-D signaling pathways-Reference-Cited by-同舟云学术

Protection of pancreatic β-cell by phosphocreatine through mitochondrial improvement via the regulation of dual AKT/IRS-1/GSK-3β and STAT3/Cyp-D signaling pathways

Published:2021-08-28 Issue: Volume: Page:
ISSN:0742-2091
Container-title:Cell Biology and Toxicology
language:en
Short-container-title:Cell Biol Toxicol

Author:

Wang Hongyan,Ai Jie,Shopit Abdullah,Niu Mengyue,Ahmed Nisar,Tesfaldet Tsehaye,Tang Zhongyuan,Li Xiaodong,Jamalat Yazeed,Chu Peng,Peng Jinyong,Ma Xiaodong,Qaed Eskandar,Han Guozhu,Zhang Weisheng,Wang Jun,Tang Zeyao

Funder

Innovative Research Group Project of the National Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Cell Biology,Toxicology

Link

https://link.springer.com/content/pdf/10.1007/s10565-021-09644-7.pdf

Reference45 articles.

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3. Archuleta TL, Lemieux AM, Saengsirisuwan V, Teachey MK, Lindborg KA, Kim JS, Henriksen EJ. Oxidant stress-induced loss of IRS-1 and IRS-2 proteins in rat skeletal muscle: role of p38 MAPK. Free Radical Biol Med. 2009;47:1486–93.

4. Bartos V, Jirkovska A, Koznarova R. Risk factors for diabetic foot in recipients of renal and pancreatic transplants. Casopis Lekaru Ceskych. 1997;136:527–9.

5. Bensellam M, Jonas JC, Laybutt DR. Mechanisms of beta-cell dedifferentiation in diabetes: recent findings and future research directions. J Endocrinol. 2018;236:R109-r143.

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