Hyperglycaemia-Induced Contractile Dysfunction and Apoptosis in Cardiomyocyte-Like Pulsatile Cells Derived from Mouse Embryonic Stem Cells
Author:
Funder
South African Medical Research Council
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine,Toxicology,Molecular Biology
Link
https://link.springer.com/content/pdf/10.1007/s12012-021-09660-3.pdf
Reference42 articles.
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2. Tinker, S. C., Gilboa, S. M., Moore, C. A., Waller, D. K., Simeone, R. M., Kim, S. Y., et al. (2020). Specific birth defects in pregnancies of women with diabetes: national birth defects prevention study, 1997–2011. American Journal of Obstetrics and Gynecology, 222, 176.e1-176.e11
3. Loffredo, C. A., Wilson, P. D., & Ferencz, C. (2001). Maternal diabetes: an independent risk factor for major cardiovascular malformations with increased mortality of affected infants. Teratology, 64, 98–106
4. Floris, I., Descamps, B., Vardeu, A., Mitic, T., Posadino, A. M., Shantikumar, S., et al. (2015). Gestational diabetes mellitus impairs fetal endothelial cell functions through a mechanism involving microRNA-101 and histone methyltransferase enhancer of zester homolog-2. Arteriosclerosis, Thrombosis, and Vascular Biology, 35, 664–674
5. Kumar, S. D., Dheen, S. T., & Tay, S. S. (2007). Maternal diabetes induces congenital heart defects in mice by altering the expression of genes involved in cardiovascular development. Cardiovascular Diabetology, 6, 34
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