Central Blockade of E-Prostanoid 3 Receptor Ameliorated Hypertension Partially by Attenuating Oxidative Stress and Inflammation in the Hypothalamic Paraventricular Nucleus of Spontaneously Hypertensive Rats

Author:

Wang Fang-Fang,Ba Juan,Yu Xiao-Jing,Shi Xiao-Lian,Liu Jin-Jun,Liu Kai-Li,Fu Li-Yan,Su Qing,Li Hong-Bao,Kang Kai B.,Yi Qiu-Yue,Wang Shu-Qiu,Gao Hong-Li,Qi Jie,Li Ying,Zhu Guo-Qing,Kang Yu-Ming

Publisher

Springer Science and Business Media LLC

Subject

Cardiology and Cardiovascular Medicine,Toxicology,Molecular Biology

Reference56 articles.

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2. Shanks, J., de Morais, S. D. B., Gao, L., Zucker, I. H., & Wang, H. J. (2019). TRPV1 (transient receptor potential vanilloid 1) cardiac spinal afferents contribute to hypertension in spontaneous hypertensive rat. Hypertension, 74, 910–920.

3. Mancia, G., & Grassi, G. (2014). The autonomic nervous system and hypertension. Circulation Research, 114, 1804–1814.

4. Zhang, Z. H., Yu, Y., Wei, S. G., Nakamura, Y., Nakamura, K., & Felder, R. B. (2011). EP3 receptors mediate PGE2-induced hypothalamic paraventricular nucleus excitation and sympathetic activation. The American Journal of Physiology: Heart and Circulatory Physiology, 301, H1559-1569.

5. Ando, T., Ichijo, T., Katafuchi, T., & Hori, T. (1995). Intracerebroventricular injection of prostaglandin E2 increases splenic sympathetic nerve activity in rats. American Journal of Physiology, 269, R662-668.

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