E prostanoid receptor‐3 promotes oxidized low‐density lipoprotein‐induced human aortic smooth muscle cells inflammation-Reference-Cited by-同舟云学术

E prostanoid receptor‐3 promotes oxidized low‐density lipoprotein‐induced human aortic smooth muscle cells inflammation

Published:2022-12-28 Issue:2 Volume:10 Page:1077-1089
ISSN:2055-5822
Container-title:ESC Heart Failure
language:en
Short-container-title:ESC Heart Failure

Author:

Hu Chuang‐Jia¹²³,Wang Yan‐Wei¹,Huang Wei‐Xing⁴,Xia Yu‐Bin⁵^ORCID

Affiliation:

1. Department of Cardiology First Affiliated Hospital of Shantou University Medical College Shantou Guangdong Province China

2. Laboratory of Molecular Cardiology First Affiliated Hospital of Shantou University Medical College Shantou Guangdong Province China

3. Laboratory of Medical Molecular Imaging First Affiliated Hospital of Shantou University Medical College Shantou Guangdong Province China

4. Department of Cardiac Surgery First Affiliated Hospital of Shantou University Medical College Shantou Guangdong Province China

5. Department of Nephrology First Affiliated Hospital of Shantou University Medical College No. 57, Changping Rd Shantou 515000 Guangdong Province China

Publisher

Wiley

Subject

Cardiology and Cardiovascular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ehf2.14264

Reference46 articles.

1. Coronary artery calcium is associated with increased risk for lung and colorectal cancer in men and women: the Multi-Ethnic Study of Atherosclerosis (MESA)

2. A Novel Peptide Isolated from Microalgae Isochrysis zhanjiangensis Exhibits Anti-apoptosis and Anti-inflammation in Ox-LDL Induced HUVEC to Improve Atherosclerosis

3. Endothelial-Derived APT1-Mediated Macrophage-Endothelial Cell Interactions Participate in the Development of Atherosclerosis by Regulating the Ras/MAPK Signaling Pathway

4. Cross-effect of TRPV1 and EP3 receptor on coughs and bronchopulmonary C-neural activities

5. Systemic Metabolic Alterations Correlate with Islet-Level Prostaglandin E2 Production and Signaling Mechanisms That Predict β-Cell Dysfunction in a Mouse Model of Type 2 Diabetes

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