Down-regulation of SETD6 protects podocyte against high glucose and palmitic acid-induced apoptosis, and mitochondrial dysfunction via activating Nrf2-Keap1 signaling pathway in diabetic nephropathy-Reference-Cited by-同舟云学术

Down-regulation of SETD6 protects podocyte against high glucose and palmitic acid-induced apoptosis, and mitochondrial dysfunction via activating Nrf2-Keap1 signaling pathway in diabetic nephropathy

Published:2020-08-16 Issue:5 Volume:51 Page:549-558
ISSN:1567-2379
Container-title:Journal of Molecular Histology
language:en
Short-container-title:J Mol Hist

Author:

Wang Xiang,Liu Qiling,Kong Deqin,Long Zi,Guo YuFang,Wang Shuang,Liu Rui,Hai Chunxu

Funder

Shaanxi Provincial Health and Family Planning Commission Scientific Research Fund

National Natural Sciences Foundation of China

Shaanxi Province Key Research and Development Project fund

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,General Medicine,Physiology,Histology

Link

https://link.springer.com/content/pdf/10.1007/s10735-020-09904-6.pdf

Reference43 articles.

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2. Ahmad J (2015) Management of diabetic nephropathy: recent progress and future perspective. Diabetes Metab Syndr Clin Res Rev 9(4):343–358

3. Belinda J, Mythili G, Andi Q, Ying F, Chuang PY, Cohen HW, Maria A, Thomas DB, He JC (2012) Dysregulated nephrin in diabetic nephropathy of type 2 diabetes: a cross sectional study. PLoS ONE 7(5):e36041

4. Binda O, Sevilla A, LeRoy G, Lemischka IR, Garcia BA, Richard S (2013) SETD6 monomethylates H2AZ on lysine 7 and is required for the maintenance of embryonic stem cell self-renewal. Epigenetics 8(2):177–183

5. Bommer J (2001) Attaining long-term survival when treating diabetic patients with ESRD by hemodialysis. Adv Ren Replace Ther 8(1):13–21

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