Lipopolysaccharide-Elicited Systemic Inflammation Induces Selective Vulnerability of Cerebral Cortex and Striatum of Developing Glutaryl-CoA Dehydrogenase Deficient (Gcdh−/−) Mice to Oxidative Stress-Reference-Cited by-同舟云学术

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Lipopolysaccharide-Elicited Systemic Inflammation Induces Selective Vulnerability of Cerebral Cortex and Striatum of Developing Glutaryl-CoA Dehydrogenase Deficient (Gcdh−/−) Mice to Oxidative Stress

Published:2020-10-01 Issue:4 Volume:38 Page:1024-1036
ISSN:1029-8428
Container-title:Neurotoxicity Research
language:en
Short-container-title:Neurotox Res

Author:

Seminotti Bianca^ORCID,Amaral Alexandre Umpierrez^ORCID,Grings Mateus^ORCID,Ribeiro César Augusto João^ORCID,Leipnitz Guilhian^ORCID,Wajner Moacir^ORCID

Funder

Conselho Nacional de Desenvolvimento Científico e Tecnológico

Fundação de Amparo à Pesquisa do Estado do Rio Grande do Sul

Hospital de Clínicas de Porto Alegre

Financiadora de Estudos e Projetos

Instituto Nacional de Ciência e Tecnologia para Excitotoxicidade e Neuroproteção

Coordenação de Aperfeiçoamento de Pessoal de Nível Superior

Publisher

Springer Science and Business Media LLC

Subject

Toxicology,General Neuroscience

Link

https://link.springer.com/content/pdf/10.1007/s12640-020-00291-0.pdf

Reference78 articles.

1. Adami C, Bianchi R, Pula G, Donato R (2004) S100B-stimulated NO production by BV-2 microglia is independent of RAGE transducing activity but dependent on RAGE extracellular domain. Biochim Biophys Acta 1742:169–177. https://doi.org/10.1016/j.bbamcr.2004.09.008

2. Amaral AU, Seminotti B, Cecatto C, Fernandes CG, Busanello EN, Zanatta A, Kist LW, Bogo MR, de Souza DO, Woontner M, Goodman S, Koeller DM, Wajner M (2012a) Reduction of Na+, K+-ATPase activity and expression in cere- bral cortex of glutaryl-CoA dehydrogenase deficient mice: a possible mechanism for brain injury in glutaric aciduria type I. Mol Genet Metab 107:375–382. https://doi.org/10.1016/j.ymgme.2012.08.016

3. Amaral AU, Cecatto C, Seminotti B, Zanatta A, Fernandes CG, Busanello EN, Braga LM, Ribeiro CA, de Souza DO, Woontner M, Koeller DM, Goodman S, Wajner M (2012b) Marked reduction of Na(+), K(+)-ATPase and creatine kinase activities induced by acute lysine administration in glutaryl-CoA dehydrogenase deficient mice. Mol Genet Metab 107:81–86. https://doi.org/10.1016/j.ymgme.2012.04.015

4. Amaral AU, Cecatto C, Seminotti B, Ribeiro CA, Lagranha VL, Pereira CC, de Oliveira FH, de Souza DG, Goodman S, Woontner M, Wajner M (2015) Experimental evidence that bioenergetics disruption is not mainly involved in the brain injury of glutaryl-CoA dehydrogenase deficient mice submitted to lysine overload. Brain Res 1620:116–129. https://doi.org/10.1016/j.brainres.2015.05.013

5. Amaral AU, Seminotti B, da Silva JC, de Oliveira FH, Ribeiro RT, Vargas CR, Leipnitz G, Santamaría A, Souza DO, Wajner M (2018) Induction of neuroinflammatory response and histopathological alterations caused by quinolinic acid administration in the striatum of glutaryl-CoA dehydrogenase deficient mice. Neurotox Res 33:593–606. https://doi.org/10.1007/s12640-017-9848-0

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2. Two novel compound heterozygous variants of the GCDH gene in two Chinese families with glutaric acidaemia type I identified by high-throughput sequencing and a literature review;Molecular Genetics and Genomics;2023-03-11

3. Disturbance of Mitochondrial Dynamics, Endoplasmic Reticulum–Mitochondria Crosstalk, Redox Homeostasis, and Inflammatory Response in the Brain of Glutaryl-CoA Dehydrogenase-Deficient Mice: Neuroprotective Effects of Bezafibrate;Molecular Neurobiology;2022-05-31

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