Induction of Neuroinflammatory Response and Histopathological Alterations Caused by Quinolinic Acid Administration in the Striatum of Glutaryl-CoA Dehydrogenase Deficient Mice-Reference-Cited by-同舟云学术

Induction of Neuroinflammatory Response and Histopathological Alterations Caused by Quinolinic Acid Administration in the Striatum of Glutaryl-CoA Dehydrogenase Deficient Mice

Published:2017-12-12 Issue:3 Volume:33 Page:593-606
ISSN:1029-8428
Container-title:Neurotoxicity Research
language:en
Short-container-title:Neurotox Res

Author:

Amaral Alexandre Umpierrez,Seminotti Bianca,da Silva Janaína Camacho,de Oliveira Francine Hehn,Ribeiro Rafael Teixeira,Vargas Carmen Regla,Leipnitz Guilhian,Santamaría Abel,Souza Diogo Onofre,Wajner Moacir^ORCID

Funder

Conselho Nacional de Desenvolvimento Científico e Tecnológico

Fundação de Amparo à Pesquisa do Estado do Rio Grande do Sul

Hospital de Clínicas de Porto Alegre

Pró-Reitoria de Pesquisa, Universidade Federal do Rio Grande do Sul

Financiadora de Estudos e Projetos

Instituto Nacional de Ciência e Tecnologia em Excitotoxicidade e Neuroproteção

Publisher

Springer Science and Business Media LLC

Subject

Toxicology,General Neuroscience

Link

http://link.springer.com/article/10.1007/s12640-017-9848-0/fulltext.html

Reference67 articles.

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2. Amaral AU, Seminotti B, Cecatto C, Fernandes CG, Busanello EN, Zanatta A, Kist LW, Bogo MR, de Souza DO, Woontner M, Goodman S, Koeller DM, Wajner M (2012b) Reduction of Na+, K+-ATPase activity and expression in cerebral cortex of glutaryl-CoA dehydrogenase deficient mice: a possible mechanism for brain injury in glutaric aciduria type I. Mol Genet Metab 107(3):375–382. https://doi.org/10.1016/j.ymgme.2012.08.016

3. Amaral AU, Cecatto C, Seminotti B, Ribeiro CA, Lagranha VL, Pereira CC, de Oliveira FH, de Souza DG, Goodman S, Woontner M, Wajner M (2015) Experimental evidence that bioenergetics disruption is not mainly involved in the brain injury of glutaryl-CoA dehydrogenase deficient mice submitted to lysine overload. Brain Res 1620:116–129. https://doi.org/10.1016/j.brainres.2015.05.013

4. Boy N, Muhlhausen C, Maier EM, Heringer J, Assmann B, Burgard P, Dixon M, Fleissner S, Greenberg CR, Harting I, Hoffmann GF, Karall D, Koeller DM, Krawinkel MB, Okun JG, Opladen T, Posset R, Sahm K, Zschocke J, Kolker S, Additional individual c (2017) Proposed recommendations for diagnosing and managing individuals with glutaric aciduria type I: second revision. J Inherit Metab Dis 40(1):75–101. https://doi.org/10.1007/s10545-016-9999-9

5. Brismar J, Ozand PT (1995) CT and MR of the brain in glutaric acidemia type I: a review of 59 published cases and a report of 5 new patients. AJNR Am J Neuroradiol 16(4):675–683

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2. Increased susceptibility to quinolinic acid‐induced seizures and long‐term changes in brain oscillations in an animal model of glutaric acidemia type I;Journal of Neuroscience Research;2021-10-28

3. Lipopolysaccharide-Elicited Systemic Inflammation Induces Selective Vulnerability of Cerebral Cortex and Striatum of Developing Glutaryl-CoA Dehydrogenase Deficient (Gcdh−/−) Mice to Oxidative Stress;Neurotoxicity Research;2020-10-01

4. Acute lysine overload provokes marked striatum injury involving oxidative stress signaling pathways in glutaryl-CoA dehydrogenase deficient mice;Neurochemistry International;2019-10

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