BMAA, Neurodegeneration, and Neuroprotection
Author:
Publisher
Springer Science and Business Media LLC
Subject
Toxicology,General Neuroscience
Link
http://link.springer.com/content/pdf/10.1007/s12640-020-00303-z.pdf
Reference42 articles.
1. Andersson M, Karlsson O, Bergström U, Brittebo EB, Brandt I (2013) Maternal transfer of the cyanobacterial neurotoxin β-N-methylamino-L-alanine (BMAA) via milk to suckling offspring. PLoS One 8(10):e78133. https://doi.org/10.1371/journal.pone.0078133
2. Andersson M, Ersson L, Brandt I, Bergström U (2017) Potential transfer of neurotoxic amino acid β-N-methylamino-alanine (BMAA) from mother to infant during breast-feeding: predictions from human cell lines. Toxicol Appl Pharmacol 320:40–50
3. Banack SA (2020) Second laboratory validation for the of β-N-methylamino-L-alanine, N-(2aminoethyl) glycine, and 2,4-diaminobuytric acid by ultra-performance liquid chromatography and tandem mass spectrometry. Neurotox Res 38(4) in press
4. Beach DG, Kerrin ES, Quilliam MA (2015) Selective quantitation of the neurotoxin BMAA by use of hydrophilic-interaction liquid chromatography–differential mobility spectrometry–tandem mass 335 spectrometry (HILIC–DMS–MS/MS). Anal Bioanal Chem 407(28):8397–8409
5. Bell EA (2009) The discovery of BMAA, and examples of biomagnification and protein incorporation involving other non-protein amino acids. Amyotroph Lateral Scler 10(sup2):21–25. https://doi.org/10.3109/17482960903268700
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