Doxorubicin Cardiotoxicity: Pathophysiology Updates
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine
Link
https://link.springer.com/content/pdf/10.1007/s11936-020-00842-w.pdf
Reference94 articles.
1. • Cardinale D, Colombo A, Bacchiani G, et al. Early detection of anthracycline cardiotoxicity and improvement with heart failure therapy. Circulation. 2015;131(22):1981–8. https://doi.org/10.1161/CIRCULATIONAHA.114.013777. This is a study of the change in LVEF in a cohort of 2625 patients receiving chemotherapy with anthrayclines. Cardiotoxicity developed at a mean of 3.5 months after completion of chemotherapy, and 98% of cardiotoxicity was seen within the first year.
2. von Hoff DD, Layard MW, Basa P, Davis HL Jr, von Hoff A, Rozencweig M, et al. Risk factors for doxorubicin-induced congestive heart failure. Ann Intern Med. 1979;91(5):710–7. https://doi.org/10.7326/0003-4819-91-5-710.
3. Zamorano JL, Lancellotti P, Rodriguez Muñoz D, Aboyans V, Asteggiano R, Galderisi M, et al. 2016 ESC Position Paper on cancer treatments and cardiovascular toxicity developed under the auspices of the ESC Committee for Practice Guidelines: The Task Force for cancer treatments and cardiovascular toxicity of the European Society of Cardiology (ESC). Eur J Heart Fail. 2017;19(1):9–42. https://doi.org/10.1002/ejhf.654.
4. Reichardt P, Tabone M-D, Mora J, Morland B, Jones RL. Risk–benefit of dexrazoxane for preventing anthracycline-related cardiotoxicity: re-evaluating the European labeling. Future Oncol. 2018;14(25):2663–76. https://doi.org/10.2217/fon-2018-0210.
5. Saleme B, Gurtu V, Zhang Y, et al. Tissue-specific regulation of p53 by PKM2 is redox dependent and provides a therapeutic target for anthracycline-induced cardiotoxicity. Sci Transl Med. 2019;11(478):eaau8866. https://doi.org/10.1126/scitranslmed.aau8866.
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