N-Propargyl Caffeamide (PACA) Ameliorates Dopaminergic Neuronal Loss and Motor Dysfunctions in MPTP Mouse Model of Parkinson’s Disease and in MPP+-Induced Neurons via Promoting the Conversion of proNGF to NGF-Reference-Cited by-同舟云学术

N-Propargyl Caffeamide (PACA) Ameliorates Dopaminergic Neuronal Loss and Motor Dysfunctions in MPTP Mouse Model of Parkinson’s Disease and in MPP+-Induced Neurons via Promoting the Conversion of proNGF to NGF

Published:2017-03-21 Issue:3 Volume:55 Page:2258-2267
ISSN:0893-7648
Container-title:Molecular Neurobiology
language:en
Short-container-title:Mol Neurobiol

Author:

Luo Dan,Zhao Jia,Cheng Yuanyuan,Lee Simon Ming-Yuen,Rong Jianhui

Funder

General Research Fund (GRF) grants from the Research Grants Council of Hong Kong

Publisher

Springer Science and Business Media LLC

Subject

Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology

Link

http://link.springer.com/article/10.1007/s12035-017-0486-6/fulltext.html

Reference49 articles.

1. Brown CA, Cheng EM, Hays RD, Vassar SD, Vickrey BG (2009) SF-36 includes less Parkinson disease (PD)-targeted content but is more responsive to change than two PD-targeted health-related quality of life measures. Qual Life Res 18(9):1219–1237. doi: 10.1007/s11136-009-9530-y

2. Tieu K, Perier C, Caspersen C, Teismann P, Wu DC, Yan SD, Naini A, Vila M et al (2003) D-beta-hydroxybutyrate rescues mitochondrial respiration and mitigates features of Parkinson disease. J Clin Invest 112(6):892–901. doi: 10.1172/JCI18797

3. Jonnala RR, Buccafusco JJ (2001) Inhibition of nerve growth factor signaling by peroxynitrite. J Neurosci Res 63(1):27–34. doi: 10.1002/1097-4547(20010101)63:1<27::AID-JNR4>3.0.CO;2-#

4. Bruno MA, Cuello AC (2012) Cortical peroxynitration of nerve growth factor in aged and cognitively impaired rats. Neurobiol Aging 33(9):1927–1937. doi: 10.1016/j.neurobiolaging.2011.09.031

5. Tillerson JL, Caudle WM, Reveron ME, Miller GW (2003) Exercise induces behavioral recovery and attenuates neurochemical deficits in rodent models of Parkinson’s disease. Neuroscience 119(3):899–911. doi: 10.1016/S0306-4522(03)00096-4

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