Quantitative Phosphoproteomics Reveals Extensive Protein Phosphorylation Dysregulation in the Cerebral Cortex of Huntington’s Disease Mice Prior to Onset of Symptoms
Author:
Publisher
Springer Science and Business Media LLC
Subject
Neuroscience (miscellaneous),Cellular and Molecular Neuroscience,Neurology
Link
https://link.springer.com/content/pdf/10.1007/s12035-021-02698-y.pdf
Reference61 articles.
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3. Butterfield DA (2019) Phosphoproteomics of Alzheimer disease brain: insights into altered brain protein regulation of critical neuronal functions and their contributions to subsequent cognitive loss. Biochim Biophys Acta - Mol Basis Dis 1865:2031–2039. https://doi.org/10.1016/j.bbadis.2018.08.035
4. MacDonald ME, Ambrose CM, Duyao MP et al (1993) A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington’s disease chromosomes. Cell 72:971–983. https://doi.org/10.1016/0092-8674(93)90585-E
5. Mangiarini L, Sathasivam K, Seller M et al (1996) Exon I of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice. Cell 87:493–506. https://doi.org/10.1016/S0092-8674(00)81369-0
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