CCAAT/enhancer binding proteins play a role in oriLyt-dependent genome replication during MHV-68 de novo infection
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Drug Discovery,Biochemistry,Biotechnology
Link
http://link.springer.com/content/pdf/10.1007/s13238-011-1060-z.pdf
Reference19 articles.
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2. Deng, H., Chu, J.T., Park, N.H., and Sun, R. (2004). Identification of cis sequences required for lytic DNA replication and packaging of murine gammaherpesvirus 68. J Virol 78, 9123–9131.
3. Efstathiou, S., Ho, Y.M., Hall, S., Styles, C.J., Scott, S.D., and Gompels, U.A. (1990). Murine herpesvirus 68 is genetically related to the gammaherpesviruses Epstein-Barr virus and herpesvirus saimiri. J Gen Virol 71, 1365–1372.
4. Gong, D., Qi, J., Arumugaswami, V., Sun, R., and Deng, H. (2009). Identification and functional characterization of the left origin of lytic replication of murine gammaherpesvirus 68. Virology 387, 285–295.
5. Hu, J., and Renne, R. (2005). Characterization of the minimal replicator of Kaposi’s sarcoma-associated herpesvirus latent origin. J Virol 79, 2637–2642.
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1. Murine Gammaherpesvirus 68 ORF48 Is an RTA-Responsive Gene Product and Functions in both Viral Lytic Replication and Latency duringIn VivoInfection;Journal of Virology;2015-03-11
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