Cardiac dysrhythmia produced by Mesobuthus tamulus venom involves NO-dependent G-Cyclase signaling pathway
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s00210-008-0375-7.pdf
Reference38 articles.
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2. Alex AB, Deshpande SB (1999) Indian red scorpion venom modulates spontaneous activity of rat right atria through the involvement of cholinergic and adrenergic systems. Indian J Exp Biol 37:455–460
3. Alex AB, Kanoo S, Deshpande SB (2006) Estrogen modulates in vitro atrial bradycardia induced by Indian red scorpion venom via G-protein coupled mechanisms. Eur J Pharmacol 546:102–108
4. Cannon SD, Wilson SP, Walsh KB (1994) A G protein-activated K+ current in bovine adrenal chromaffin cells: possible regulatory role in exocytosis. Mol Pharmacol 45:109–116
5. Chen H, Heinemann SH (2001) Interaction of scorpion alpha-toxin with cardiac sodium channels: binding properties and enhancement of slow inactivation. J Gen Physiol 117:505–518
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1. Lignocaine augments the in-vitro uterine contractions involving NO-guanylyl cyclase dependent mechanisms;Life Sciences;2017-12
2. Mesobuthus tamulus venom induces acute respiratory distress syndrome in rats involving additional mechanisms as compared to oleic acid model;Toxicon;2015-04
3. The effect of bisphenol A on some oxidative stress parameters and acetylcholinesterase activity in the heart of male albino rats;Cytotechnology;2013-12-12
4. Bisphenol A decreases atrial contractility involving NO-dependent G-cyclase signaling pathway;Journal of Applied Toxicology;2011-02-23
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