Bisphenol A decreases atrial contractility involving NO-dependent G-cyclase signaling pathway

Author:

Pant Jayanti1,Ranjan Pratibha1,Deshpande Shripad B.1

Affiliation:

1. Department of Physiology; Institute of Medical Sciences; Banaras Hindu University; Varanasi; 221005; India

Publisher

Wiley

Subject

Toxicology

Reference27 articles.

1. Indian red scorpion venom modulates the spontaneous activity of rat right atria through the involvement of cholinergic and adrenergic systems;Alex;Ind. J. Exp. Biol.,1999

2. Estrogen modulates in vitro atrial bradycardia induced by Indian red scorpion venom via G-protein coupled mechanisms;Alex;Eur. J. Pharmacol.,2006

3. Exposure to the estrogenic pollutant bisphenol A affects pain behavior induced by subcutaneous formalin injection in male and female rats;Aloisi;Brain Res.,2002

4. Nitric oxide and cGMP cause vasorelaxation by activation of a charybdotoxin-sensitive K-channel by cGMP-dependent protein kinase;Archer;Proc. Natl Acad. Sci. USA,1994

5. Bisphenol A activates Maxi-K (K(Ca)1.1) channels in coronary smooth muscle;Asano;Br. J. Pharmacol.,2010

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